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Still left a standard coronary heart and proper the reworking induced by AAS use. AAS use can cause concentric still left ventricular hypertrophy, as signified by an increased LV posterior wall and interventricular septum thickness.
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-estradiol). Most other AAS usually are not a substrate for aromatase or are transformed at decreased rates, although the latter group can even now generate a substantial number of estrogen if administered in higher doses. The manufactured estrogen subsequently exerts its consequences by binding to estrogen receptors α